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Aminoguanidine, an iNOS inhibitor, was able to inhibit the formation of NO and interleukin-1-beta (IL-1) that was caused by this upregulation 147
Back Wu, AY, Stokes, R, Peebles, R (6 Sept 2021) 'The GLP-1 receptor in airway inflammation in asthma: a promising novel target?' Expert Review of Clinical Immunology , 17/10, 105357
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T cells expanded in presence of il-15 exhibit increased antioxidant capacity and innate effector molecules
This allows the study to observe the effects of the primary agent in isolation
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